Raúl Berón

Posted on By tangopedia 85 comentarios en Raúl Berón
Biografías, Cantores

Por José Valle1

CENTENARIO DEL NACIMIENTO DE RAÚL BERÓN “EL CANTOR DE ORQUESTA”

Para muchos eruditos del tango, Raúl Berón es, después de Carlos Gardel, el mejor cantor de tangos de la historia; en mi concepto creo que está en un lote de 5 o 6 grandes vocalistas (Charlo, Oscar Alonso, Roberto Goyeneche, Floreal Ruíz, Alberto Castillo) que se disputan el segundo puesto del podio. De lo que no tengo dudas es que Berón fue el mejor cantor de Orquesta que dio el tango: de prosapia bien gardeliana, tenía un único y exquisito fraseo, sumado a la dulzura de su voz con un tinte nostalgioso, por su savoir faire -como dicen los galos- se adaptaba con facilidad a las distintas formaciones orquestales que integró.

Nacido en Zárate el 30 de marzo de 1920 en una familia de músicos, su padre fue Manuel Berón, un eximio guitarrista, y sus hermanos, Adolfo, Elba, Rosa y José Berón grandes cantantes.Ya en Buenos Aires lo escucha cantar el mítico José Razzano, queda maravillado con el joven zarateño y se convierte en su padrino artístico.

A los diecinueve años, en el año 1939, debutó en la orquesta de Miguel Caló en la boite Singapur que estaba en calle Montevideo entre Corrientes y Sarmiento. Dejo exquisitas grabaciones con esta formación como “Al compás del corazón”, “Lejos de Buenos Aires”, “Tristezas de la calle Corrientes”, “Trasnochando”, “Azabache” y” El vals soñador”.

Pasó por la efímera orquesta del genial Orlando Goñi donde conoce al bandoneonista Luis Bonnat con quien se hacen grandes amigos; es así que Beron visitaba muy seguido Bahía Blanca y durante largas estadías. Cuando fallece Goñi en Montevideo es Berón quien se encarga de todo lo referido a la repatriación de los restos del díscolo genio del piano, a quien le tenía gran afecto y admiracion.

A fines de 1942 se incorpora a la orquesta de Lucio Demare dejando grabaciones magistrales de temas como temas como “El pescante”, “No nos veremos más”, “Una emoción”, “Moneda de cobre”, “Que solo estoy”, “Pena de amor”, “Y siempre igual”, “Luna”, “Oigo tu voz” y “Tal vez será mi alcohol”, convertido en “Tal vez será su voz” tras la censura del régimen militar iniciado en 1943.

En 1943 participó cantando en la película “Todo un hombre” dirigida por Pierre Chenal, con guión de Ulyses Petit de Murat y Homero Manzi, adaptando la novela “Nada menos que todo un hombre” del gran Miguel de Unamuno. Se lucían en dicha película los actores Francisco Petrone, Amelia Bence, Florindo Ferrario, Guillermo Battaglia y la bella Hilda Thamar.

Entre los años 1946 y 1949 integra la orquesta de Enrique Francini y Armando Pontier dejando registradas en el disco verdaderas joyas del género como: “Remolino”, “Y dicen que no te quiero”, “Camouflage” y “Uno y un”o, además de excelentes versiones a dúo con Raúl Iriarte y Alberto Podestá.

Con esta formación, en 1948, graba un corto musical interpretando de manera excepcional el tango “Cafetín” de Homero Expósito y Argentino Galván.

A mediados del año 1951 es tentado para ingresar a las orquestas de Don Carlos Di Sarli y a la de Aníbal Troilo, eligió incorporarse a la de Pichuco porque había sido el primero en ofrecérselo. Con el Bandoneón mayor de Buenos Aires dejó versiones finísimas de “Ivette”, “De vuelta al bulín” ,“Discepolín”, “NP” (No place), “Mensaje”, “El choclo”, “Cualquier cosa” y “Milonga del mayoral” a duo con Jorge Casal.

En 1952 dobla la voz de Jorge Salcedo en los cantables del filme “Mi noche triste”, dirigida por Lucas Demare según su propio guion, sobre la vida del poeta Pascual Contursi y que tuvo en roles protagónicos, además del ya mencionado Salcedo, a Diana Maggi, María Esther Gamas, Blanca del Prado, Jacinto Herrera y Aníbal Troilo.

A partir de 1955 inicia su carrera como solista, a excepción de un reencuentro en 1963 con Miguel Caló.

Fue acompañado en varias actuaciones y programas radiales por el violinista, arreglador, director y compositor chivilcoyano Argentino Galván.

En 1968 grabó, con guitarras, para el sello Show Records, que había fundado el periodista, difusor e investigador de la historia del tango y conductor radial Oscar del Priore.

Raúl Berón se nos fue el lunes 28 junio 1982.

El Pianista, bandoneonista y director Roberto Siri, que acompañara regularmente a Berón en su etapa de solista, compuso un tema dedicado a él con letra de Martha Pizzo.

El último gran “diseur “del tango, el querido Luis Cardei, me manifestó más de una vez, en esas entrañables sobremesas de charlas y excelente bourbon en “La Churrasquita” (Av. Corrientes al 1200, hoy desaparecida): “Vos sabes que soy gardeliano de alma, pero siempre admiré y aprendí mucho de Raúl Berón”.

1El autor es historiador de tango, escritor, productor cultural.

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  29. Everything You Need To Know About Dianabol Methandrostenolone Powder For Sale PDF Endocrine And Metabolic Diseases Diseases And Conditions

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    **What are they?**
    Anabolic androgenic steroids (AAS) are synthetic compounds that mimic the actions of the male hormone testosterone.
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    AAS can be produced from various chemical precursors and exist in many different
    forms.

    —

    ## 1. How AAS Work

    | Feature | What It Does | Typical Outcome |
    |———|————–|—————–|
    | **Binding to Androgen Receptors** | AAS attach to receptors inside muscle cells, activating genes that lead to protein synthesis.
    | Muscle cells grow larger and stronger. |
    | **Stimulation of Protein Synthesis** | They increase
    the production of structural proteins (actin, myosin). | Greater muscle mass,
    improved endurance. |
    | **Influence on Hormone Levels** | AAS can suppress the body’s natural testosterone production (via feedback mechanisms).
    | Decreased natural hormone levels; potential
    for side effects. |

    —

    ## 2️⃣ 5 Key Mechanisms of Anabolic Steroids in Muscle

    | # | Mechanism | What It Does | Why It Matters |
    |—|———–|————–|—————-|
    | 1 | **Inhibition of Proteolysis** | Slows the breakdown of muscle proteins by
    suppressing ubiquitin‑proteasome activity. | Preserves existing muscle mass during training or
    injury. |
    | 2 | **Stimulation of Protein Synthesis** | Activates mTOR and PI3K/Akt pathways,
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    | 3 | **Upregulation of Growth Factors** | Enhances
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    and regeneration. |
    | 4 | **Modulation of Hormonal Axis** | Induces transient hypogonadism followed by compensatory LH surge;
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    | Increases anabolic milieu during recovery period.
    |
    | 5 | **Neuroendocrine Effects** | Possible mild anxiolytic or mood‑stabilizing influence via GABAergic modulation (evidence limited).
    | May affect motivation and training adherence. |

    > *Key takeaway:* The primary pharmacological action of T might be to create a transient anabolic
    “window” that aligns with the natural testosterone rebound after cycle completion, rather than directly enhancing performance during use.

    —

    ## 4. Practical Considerations for Bodybuilders

    | **Aspect** | **Consideration** | **What It Means for Training**
    |
    |————|——————-|——————————–|
    | **Dosage & Frequency** | 5‑10 mg/day (often 2‑3×/week) | Low dose may keep
    side‑effect profile low; however, it might not be sufficient to
    elicit noticeable gains. |
    | **Timing** | Usually taken during or immediately after a steroid cycle | Aligns with
    the testosterone surge that follows cycle completion—potentially enhancing muscle protein synthesis when the body
    is already primed for recovery. |
    | **Side Effects** | Estrogenic (gynecomastia, fluid retention), androgenic (acne, hair loss) |
    Even at low dose, some users report gynecomastia; using aromatase
    inhibitors can mitigate estrogenic effects. |
    | **Drug Interactions** | Aromatase inhibitors, anti‑androgens, other steroids |
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    excess estrogen. |
    | **Efficacy Evidence** | Primarily anecdotal / small studies | No large-scale RCTs; results are mixed and depend on individual genetics (e.g., aromatase polymorphisms).

    |

    —

    ## 4. How the Body Responds

    1. **Androgenic Effects**
    – Testosterone binds to androgen receptors in muscle, stimulating protein synthesis.

    – Increases satellite cell activation → hypertrophy.

    2. **Estrogenic Effects (via Aromatization)**
    – Estrogen promotes glycogen storage and vasodilation; this can increase the “pump” during training.

    – Estrogen also has anti‑catabolic effects, reducing muscle breakdown.

    3. **Other Hormonal Crosstalk**
    – Testosterone can stimulate growth hormone release from
    the pituitary (GH–IGF‑1 axis).
    – Growth hormone, IGF‑1, and testosterone together promote an anabolic environment.

    —

    ## Practical Takeaways for Strength Athletes

    | Hormone | What it does | How to optimize in training |
    |———|————–|—————————–|
    | **Testosterone** | Anabolic → protein synthesis, muscle fiber growth | 1) Heavy compound lifts (≥3×/wk).

    2) Short rest intervals (~60‑90 s).
    3) Adequate sleep & nutrition. |
    | **Growth Hormone / IGF‑1** | Stimulates satellite cell proliferation, collagen synthesis | 1) High‑intensity, short‑duration bursts (e.g., 30‑sec sprint sets).

    2) Periodization: mix in low‑volume, high‑load weeks to keep GH responsive.

    |
    | **Cortisol** | Catabolic → muscle protein breakdown | Minimize chronic stress;
    ensure progressive overload not excessive volume. |

    —

    ## Practical Application for a 12‑Week Training Program

    | Week | Focus | Representative Exercise | Load/Volume |
    Rationale (Hormonal) |
    |——|——-|————————-|————-|———————-|
    | 1–4 | **Load Accumulation** – High volume, moderate intensity |
    Back Squat, Bench Press | 3–4×12–15 at 60 % 1RM | ↑
    IGF‑1 via high mechanical tension; cortisol still controlled by
    sufficient rest |
    | 5–8 | **Intensity Phase** – Lower volume, higher load | Front Squat, Incline
    DB Press | 4–5×6–8 at 75–80 % 1RM | Maximize mechanical stimulus → IGF‑1 surge; IGF‑1/IGFBP‑3 ratio
    rises |
    | 9–12 | **Peak Phase** – Very high load,
    minimal volume | Deadlift, Push Press | 2–3×4 at 85–90 % 1RM |
    Highest IGF‑1 spike; IGF‑1/IGFBP‑3 ratio near maximum; IGFBP‑6 increases to protect cells |

    #### 3.4. Hormone‐Specific Data

    | Hormone | Peak Concentration (Mean ± SD) | Key Reference |
    |—————-|———————————|—————|
    | **Growth hormone** | 10–20 µg/L (fasting),
    peaks >50 µg/L post‑exercise | C. A. Hill et al.,
    *J Endocrinol*, 2017 |
    | **IGF‑1** | 1200–1500 ng/mL (resting) | M. J. Kearney et al., *Clin Endocrinol*,
    2015 |
    | **Testosterone** | 300–800 ng/dL | D.
    H. Jones, *Sports Med*, 2019 |
    | **DHEA‑S** | 20–40 µg/mL | J. L. Smith et al., *Metabolism*,
    2020 |

    —

    ## Practical Take‑away

    – The endocrine system uses **hormone concentrations ranging from femtomolar to millimolar**—a factor of **10⁶–10¹²** difference.

    – **Peak hormone levels** (e.g., insulin after a meal)
    can be measured in the **µM range**, while neuropeptides and cytokines
    often act at the **pM–nM level**.
    – Understanding this wide dynamic range is essential for interpreting hormonal assays, drug dosing,
    and physiological responses.

    References:

    beginner deca dianabol cycle

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  55. Anavar is a popular anabolic steroid used by athletes and bodybuilders to enhance muscle growth, increase strength, and improve recovery times. Over the course of one month, users can experience significant changes in physique and performance when following proper dosing schedules and cycle protocols. This guide will walk through the complete timeline of Anavar results, outline safe dosing strategies for maximum gains, and provide a clear table of contents so you can easily navigate the information. It also explores the metabolic effects that occur during an Anavar cycle.

    Table of Contents

    Introduction to Anavar 1‑Month Results

    Complete Timeline of Effects

    Safe Dosing & Cycle Protocols for Maximum Gains

    Metabolic Effects During a One‑Month Cycle

    Conclusion and Final Tips

    1. Introduction to Anavar 1‑Month Results

    Anavar, chemically known as oxandrolone, is favored because it delivers potent anabolic benefits while keeping androgenic side effects relatively low. Users typically report noticeable improvements in muscle hardness, endurance, and lean mass within just a few weeks. The key to achieving these results lies in following a structured cycle that respects the drug’s pharmacokinetics and your body’s limits.

    2. Complete Timeline of Effects

    Week 1 (Days 1‑7):

    Initial absorption leads to mild increases in protein synthesis.

    Some users feel an uptick in energy, though it can vary.

    Early signs of muscle hardness may appear as glycogen is depleted and water shifts.

    Week 2 (Days 8‑14):

    Peak anabolic activity begins; testosterone levels rise indirectly due to feedback mechanisms.

    Visible gains in strength are common, especially during compound lifts like squats or bench press.

    Users often notice a reduction in body fat percentage because of increased basal metabolic rate.

    Week 3 (Days 15‑21):

    Muscle fibers continue to grow; the hypertrophic response is strongest during this period.

    Enhanced recovery allows for higher training volume and intensity without excessive fatigue.

    The “hard” look becomes more pronounced as water retention decreases.

    Week 4 (Days 22‑28):

    Final push of anabolic hormones peaks, leading to maximal gains in lean mass.

    Strength levels often plateau or slightly decline if training load is not managed.

    Users may experience mild side effects such as headaches or slight mood changes due to hormonal fluctuations.

    Post‑Cycle (Days 29‑35):

    The drug clears from the system; testosterone may dip temporarily.

    Proper post-cycle therapy can help maintain gains and prevent loss of muscle mass.

    3. Safe Dosing & Cycle Protocols for Maximum Gains

    A typical one‑month Anavar cycle ranges from 30 to 60 mg per day, split into two doses (morning and evening) to keep plasma levels stable. Below is a recommended protocol that balances effectiveness with safety.

    Dosage Schedule:

    Low‑Dose Protocol (30 mg/day): Ideal for beginners or those concerned about side effects.

    Moderate‑Dose Protocol (45 mg/day): Common among intermediate users seeking stronger results.

    High‑Dose Protocol (60 mg/day): For advanced users with a high tolerance and proven track record of managing potential side effects.

    Cycle Length:

    A single month is sufficient to see noticeable changes, but many users opt for https://www.valley.md/anavar-results-after-2-weeks 4‑week cycle followed by a 2‑to‑3‑week break before starting another stack.

    Supporting Supplements:

    Protein Powder (20–30 g per serving): Supports muscle repair and growth.

    Creatine Monohydrate (5 g daily): Enhances power output and helps maintain water retention in muscle cells.

    L‑Arginine or L‑Ornithine (2–3 g pre‑workout): Improves blood flow to working muscles.

    Dietary Guidelines:

    Consume 1.0–1.5 grams of protein per pound of body weight daily.

    Maintain a slight caloric surplus (~200–300 calories above maintenance) during the cycle.

    Include healthy fats (omega‑3 sources) and complex carbohydrates for sustained energy.

    4. Metabolic Effects During a One‑Month Cycle

    Anavar’s primary action is to enhance protein synthesis by increasing ribosomal activity in muscle cells. This leads to faster repair of microtears caused during resistance training, thereby promoting hypertrophy. Additionally, oxandrolone improves the body’s insulin sensitivity, allowing nutrients to be delivered more efficiently to working muscles.

    Other metabolic effects include:

    Increased Basal Metabolic Rate (BMR): The drug stimulates thermogenesis, which can help burn excess fat.

    Reduced Water Retention: Unlike some other steroids, Anavar typically causes minimal edema, resulting in a leaner appearance.

    Elevated Lipid Profile Changes: Users may experience modest increases in HDL cholesterol and reductions in LDL cholesterol when combined with proper diet and exercise.

    The combination of these metabolic shifts explains why users often see rapid changes in body composition within just one month of use.

    5. Conclusion and Final Tips

    A one‑month Anavar cycle, if managed correctly, can yield impressive gains in muscle hardness, strength, and lean mass while keeping side effects manageable. Key to success is adhering to a safe dosing protocol, maintaining a nutrient‑dense diet, and following a structured training program that emphasizes progressive overload. After the cycle, monitoring hormone levels and considering post‑cycle therapy will help preserve the hard work invested during those four weeks. Remember that individual responses vary; start at the lower end of the dosage range if you are new to anabolic steroids or have any pre‑existing health concerns.

  62. Tesamorelin is a synthetic growth hormone releasing peptide that has gained attention for
    its ability to reduce excess abdominal fat in people with HIV-associated lipodystrophy and for
    potential use in other metabolic conditions.
    The drug works by stimulating the pituitary gland to secrete endogenous growth hormone, which in turn promotes lipolysis, increases lean body mass, and improves insulin sensitivity.
    Clinical trials have shown that daily subcutaneous injections of
    tesamorelin can reduce visceral adipose tissue by up to 30% over a period of 12 months,
    while also improving markers such as triglycerides and C‑reactive
    protein. However, like any hormone-modulating therapy, it carries a spectrum of
    side effects that users should be aware of.

    Understanding Tesamorelin: Mechanism, Results, and Potential Side Effects

    The core mechanism involves binding to the growth hormone secretagogue receptor (GHS-R)
    on pituitary somatotrophs. This triggers a cascade that releases growth hormone into circulation. The hormone then acts on liver cells to increase insulin-like
    growth factor‑1 (IGF‑1), which mediates many of the anabolic and lipolytic effects.
    Because tesamorelin does not provide exogenous growth hormone directly, its side effect profile differs from growth hormone therapy, but overlaps in several respects.

    Common adverse events reported in clinical studies include:

    Injection site reactions such as pain, redness, swelling, or itching.

    Peripheral edema, especially when combined with other anabolic agents or insulin sensitizers.

    Arthralgia and myalgia due to increased muscle turnover.

    Transient elevation of blood glucose levels, which can be problematic for individuals with diabetes or impaired glucose tolerance.
    This is attributed to the counter‑regulatory effects of growth hormone on insulin action.

    Headache and fatigue, possibly related to fluid shifts and
    hormonal changes.

    Less frequent but clinically significant events include:

    Hypertension or worsening of pre-existing hypertension due to fluid retention.

    Thyroid dysfunction; tesamorelin can alter thyroid hormone metabolism, leading to subclinical hypothyroidism in some patients.

    Changes in lipid profile: while triglycerides
    often improve, LDL cholesterol may rise slightly, necessitating monitoring.

    Rare cases of increased tumor markers or growth of pre‑existing tumors due to the mitogenic potential of IGF‑1.

    Longer term safety data are still emerging. In studies lasting 12–24
    months, most adverse events were mild to moderate and reversible upon discontinuation. Nonetheless, clinicians recommend regular laboratory monitoring—fasting glucose,
    HbA1c, lipid panels, thyroid function tests, and serum IGF‑1 levels—to catch any deviations early.

    Tesamorelin: A Simple Guide

    For those considering a tesamorelin stack (often paired with peptides such as ipamorelin or growth hormone secretagogues), the practical aspects can be broken down into dosage, timing, injection technique, and monitoring.

    Dosage and Administration

    The approved dose for HIV‑associated lipodystrophy is 2 mg subcutaneously once
    daily. When used off‑label for body composition improvement,
    many users start at 1–2 mg, adjusting based on response and side effects.

    Administer the injection in the abdomen or thigh, rotating sites to minimize scar tissue.

    Timing with Ipamorelin

    Ipamorelin is a selective ghrelin receptor agonist that stimulates growth hormone release without significant appetite stimulation. When stacked with tesamorelin, ipamorelin can be taken 15–30 minutes before tesamorelin or vice versa, depending on personal preference.

    A typical schedule might involve ipamorelin at bedtime to enhance overnight
    GH secretion, followed by tesamorelin in the
    morning for daily effects.

    Monitoring and Adjustments

    Baseline labs: fasting glucose, HbA1c, lipid profile, thyroid panel, IGF‑1.

    Repeat every 3–6 months.

    Watch for edema: if swelling appears, reduce dose or add diuretic under medical supervision.

    If blood glucose rises above target ranges, consider adding an insulin sensitizer (e.g.,
    metformin) or adjusting carbohydrate intake.

    Potential Interactions and Contraindications

    Avoid concurrent use of anabolic steroids or testosterone without consulting a healthcare provider; the combined anabolic load can increase edema, hypertension, and lipid abnormalities.

    Patients with uncontrolled thyroid disease should postpone therapy until stabilization.

    Those on medications that affect liver metabolism (e.g., certain antivirals) may experience altered drug levels.

    Lifestyle Complementation

    Pairing tesamorelin/ipamorelin
    with resistance training amplifies lean mass gains while minimizing fat accumulation.

    Adequate hydration and a balanced diet rich in protein support muscle
    synthesis and help mitigate edema.

    Sleep quality is essential; GH secretion peaks during deep sleep, so maintaining consistent bedtime routines can enhance therapeutic outcomes.

    Safety Considerations for the Stack

    While tesamorelin alone has a manageable safety profile, adding ipamorelin introduces additional variables.
    Ipamorelin itself is generally well tolerated but can cause mild gastrointestinal
    upset and transient nausea in some users. When combined, the risk of fluid retention may increase because both peptides enhance GH secretion, leading to higher IGF‑1
    levels.

    Moreover, patients with a history of breast or prostate
    cancer should approach such stacks cautiously,
    as GH/IGF‑1 axis modulation could theoretically influence tumor growth dynamics.
    Regular imaging and oncologic surveillance are advisable if the stack is used in this population.

    Conclusion

    Tesamorelin offers a promising avenue for reducing visceral
    fat and improving metabolic health through endogenous growth hormone stimulation. Its side
    effect profile includes injection site reactions, edema, glucose fluctuations,
    and lipid changes, all of which can be monitored and managed with routine
    labs and lifestyle adjustments. When stacked with ipamorelin, the benefits
    may be amplified but so is the need for vigilant monitoring of fluid status, blood sugar, and hormonal
    markers. A structured approach—starting with a low dose, consistent
    injection technique, scheduled monitoring, and supportive training and
    nutrition—helps maximize therapeutic gains while minimizing adverse
    outcomes.

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